Chen-Ching Yuan Email: email@example.com Structural and functional studies on animal models of hypertrophic, dilated and restrictive cardiomyopathy caused by myosin light chains mutations. Supported by AHA 15PRE23020006 Cardiomyopathy is a disease of the myocardium that could be either acquired or inherited (genetic). This study is focused on three types of inherited cardiomyopathy: hypertrophic (HCM), dilated (DCM) […]
Zhiqun Zhou, PhD Email: firstname.lastname@example.org Molecular mechanisms underlying malignant MYL2 mutations leading to cardiac and slow skeletal muscle myopathies. Supported by AHA 15POST25080302 (07/01/2015-06/30/2017) Cardioskeletal myopathy with onset and death in infancy, morphological features of muscle type I hypotrophy with myofibrillar disorganization and hypertrophic/dilated cardiomyopathy were previously reported in three Dutch families. Recently, Weterman et al. […]
Jingsheng Liang, MD Email: email@example.com Studies of the effect of genetic mutations in MYL2 and MYL3 on the skinned fiber mechanics (force-pCa, maximal tension, passive tension).
Katarzyna Kazmierczak, PhD Email: firstname.lastname@example.org Development of physiological versus pathological cardiac hypertrophy in animal models of cardiomyopathy expressing mutations in MYL2 and MYL3.
Danuta Szczesna-Cordary, PhD Email: email@example.com Myosin Light Chain Mutant Induced Cardiomyopathies The major theme of my research regards myosin and troponin-mediated cellular events that result in sarcomere shortening and muscle contraction studied at the molecular, myofilament and organ levels. Our recent research interests concentrate on the molecular and genetic causes of cardiomyopathy with a special […]
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