Zhiqun Zhou, PhD Email: firstname.lastname@example.org Molecular mechanisms underlying malignant MYL2 mutations leading to cardiac and slow skeletal muscle myopathies. Supported by AHA 15POST25080302 (07/01/2015-06/30/2017) Cardioskeletal myopathy with onset and death in infancy, morphological features of muscle type I hypotrophy with myofibrillar disorganization and hypertrophic/dilated cardiomyopathy were previously reported in three Dutch families. Recently, Weterman et al. […]
Jingsheng Liang, MD Email: email@example.com Studies of the effect of genetic mutations in MYL2 and MYL3 on the skinned fiber mechanics (force-pCa, maximal tension, passive tension).
Katarzyna Kazmierczak, PhD Email: firstname.lastname@example.org Development of physiological versus pathological cardiac hypertrophy in animal models of cardiomyopathy expressing mutations in MYL2 and MYL3.
Danuta Szczesna-Cordary, PhD Email: email@example.com Myosin Light Chain Mutant Induced Cardiomyopathies The major theme of my research regards myosin and troponin-mediated cellular events that result in sarcomere shortening and muscle contraction studied at the molecular, myofilament and organ levels. Our recent research interests concentrate on the molecular and genetic causes of cardiomyopathy with a special […]
Our new website (this one!!) is now online! 🙂